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Additionally notable in these two PD preclinical studies was an exenatide-mediated reduction in L-3,4-dihydroxyphenylalanine (L-DOPA)-induced abnormal involuntary movements (AIMs) in the rat 6-OHDA model and a slowing of disease progression, dopamine loss, and motor deficits in the MitoPark mouse when administered a clinically translatable drug dose (231)
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