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In particular, it activates pro-opiomelanocortin (POMC) neurons which trigger α-melanocyte-stimulating hormone release and melanocortin-4 receptor signaling to decrease food consumption [25,26]

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While such compounds have been described, none have yet been considered credible pharmacological candidates (253, 254) due to a combination of factors, including insufficient potency, lack of specificity of binding to the GLP-1R and suboptimal half-life