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These costs could instead be redirected to deliver new, increasingly effective treatments like GLP-1RAs to those most in need, reducing the burden of substance abuse for communities across the country.
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In a rotenone-induced PD model in rodents, DA4-JC also alleviated motor symptom dysfunctions, protecting dopaminergic neurons mediated through the restoration of mitochondrial function via the Akt/c-Jun N-terminal kinase (JNK) signaling pathway [116]
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